Saturday, December 27, 2025

Prostate Adenocarcinoma: Anatomy, Epidemiology, Etiology, Pathogenesis, Staging, Pathology, Diagnosis, Treatment and Prevention

Prostate Adenocarcinoma

~Introduction


Prostate adenocarcinoma is the most common malignant tumor of the prostate gland and one of the leading causes of cancer-related death in elderly men worldwide. It arises from the glandular epithelial cells of the prostate, predominantly from the peripheral zone. The disease typically affects men over the age of 50 years, with incidence increasing sharply with advancing age.

Most prostate cancers are slow-growing, but a subset behaves aggressively, leading to local invasion, metastasis, and death. Early diagnosis through prostate-specific antigen (PSA) testing, digital rectal examination (DRE), and biopsy has significantly improved detection rates.

~Anatomy of the Prostate

The prostate is a fibromuscular gland located below the urinary bladder and surrounds the proximal urethra.

Zones of the Prostate

  • Peripheral zone – site of ~70% of carcinomas

  • Central zone

  • Transition zone – commonly involved in benign prostatic hyperplasia (BPH)

  • Anterior fibromuscular stroma

~Epidemiology

  • Most common cancer in men (excluding skin cancer)

  • Incidence increases after 50 years

  • More common in African-American men

  • Rare before age 40

  • High prevalence in Western countries

~Etiology and Risk Factors

Age

  • Strongest risk factor

  • Majority of cases occur after 65 years

Genetic Factors

  • Family history increases risk

  • Mutations in:

    • BRCA1 and BRCA2

    • HOXB13

Hormonal Factors

  • Androgen-dependent tumor

  • Testosterone and dihydrotestosterone (DHT) play a key role

Environmental and Lifestyle Factors

  • High-fat diet

  • Obesity

  • Smoking

  • Sedentary lifestyle

~Pathogenesis

Prostate adenocarcinoma develops through a sequence of molecular and histological changes.

Precursor Lesion

  • Prostatic Intraepithelial Neoplasia (PIN)

    • Especially high-grade PIN

    • Characterized by cellular atypia without stromal invasion

Molecular Changes

  • TMPRSS2-ERG gene fusion

  • PTEN tumor suppressor gene loss

  • Increased androgen receptor signaling

~Clinical Features

Early Stage

  • Often asymptomatic

  • Detected incidentally through PSA screening

Late Stage

  • Increased urinary frequency

  • Nocturia

  • Hesitancy

  • Weak urinary stream

  • Hematuria

  • Bone pain (due to metastasis)

  • Weight loss and fatigue

~Physical Examination

Digital Rectal Examination (DRE)

  • Hard, irregular, nodular prostate

  • Loss of normal median sulcus

  • Asymmetrical enlargement

~Gross Pathology

  • Firm, ill-defined gray-white areas

  • Usually located in the peripheral zone

  • Tumor may be multifocal

  • Advanced tumors invade seminal vesicles and bladder neck

~Histopathology

Microscopic Features

  • Small, crowded malignant glands

  • Infiltration into surrounding stroma

  • Loss of basal cell layer

  • Enlarged nuclei with prominent nucleoli

  • Perineural invasion (common)

~Gleason Grading System

The Gleason score is the most important prognostic factor.

Scoring

  • Based on glandular architecture

  • Two most common patterns graded from 1 to 5

  • Gleason score = Primary grade + Secondary grade

Risk Groups

  • Score ≤6: Low grade

  • Score 7: Intermediate grade

  • Score 8–10: High grade

~TNM Staging

Tumor (T)

  • T1: Clinically inapparent

  • T2: Confined to prostate

  • T3: Extraprostatic extension

  • T4: Invades adjacent structures

Node (N)

  • N0: No lymph node involvement

  • N1: Pelvic lymph node metastasis

Metastasis (M)

  • M0: No distant metastasis

  • M1: Distant metastasis (commonly bone)

~Patterns of Spread

Local Spread

  • Seminal vesicles

  • Bladder neck

  • Urethra

Lymphatic Spread

  • Pelvic lymph nodes

  • Obturator and iliac nodes

Hematogenous Spread

  • Bones (vertebrae, pelvis, ribs)

  • Produces osteoblastic lesions

~Diagnosis

Laboratory Investigations

  • Elevated PSA

  • PSA velocity and PSA density

Imaging

  • Transrectal ultrasound (TRUS)

  • MRI prostate

  • Bone scan for metastasis

Biopsy

  • TRUS-guided core needle biopsy

  • Gold standard for diagnosis

~Differential Diagnosis

  • Benign prostatic hyperplasia (BPH)

  • Prostatitis

  • Prostatic intraepithelial neoplasia

  • Small cell carcinoma of prostate

~Treatment

Localized Disease

  • Radical prostatectomy

  • Radiation therapy (external beam or brachytherapy)

  • Active surveillance in low-risk cases

Advanced Disease

  • Androgen deprivation therapy (ADT)

  • Orchiectomy

  • Anti-androgens (bicalutamide)

  • Chemotherapy (docetaxel)

Metastatic Disease

  • Hormonal therapy

  • Palliative radiotherapy

  • Bone-targeted therapy (bisphosphonates)

~Prognosis

Prognosis depends on:

  • Gleason score

  • PSA level

  • Tumor stage

  • Lymph node involvement

Survival Rates

  • Localized disease: Excellent prognosis

  • Advanced disease: Poorer outcomes

~Prevention and Screening

  • PSA screening (controversial but useful)

  • Healthy diet

  • Regular physical activity

  • Genetic counseling in high-risk individuals

~Complications

  • Urinary incontinence

  • Erectile dysfunction

  • Bone fractures

  • Metastatic pain

  • Hormonal therapy side effects

~Conclusion

Prostate adenocarcinoma is a common malignancy of aging men with a wide spectrum of clinical behavior. Early detection through PSA testing and biopsy allows curative treatment in localized disease, while advanced stages require systemic therapy. Understanding the pathology, grading, staging, and treatment options is essential for effective management and improved patient outcomes.


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